Oh, Sehyun <br/><br/>
Human LIGIV is synthetically lethal with the loss of Rad54B-dependent recombination and is required for certain chromosome fusion events induced by telomere dysfunction.  [electronic resource] 

 -  Nucleic acids research  Feb 2013 
 - 1734-49 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1362-4962 
<br/><br/><label>Standard No.: </label>10.1093/nar/gks1326  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Antigens, Nuclear--genetics<br/>Cell Line, Tumor<br/>Cell Proliferation<br/>Chromatids<br/>Chromosome Aberrations<br/>DNA Damage<br/>DNA End-Joining Repair<br/>DNA Helicases--genetics<br/>DNA Ligase ATP<br/>DNA Ligases--genetics<br/>DNA-Binding Proteins--genetics<br/>Gene Targeting<br/>Genomic Instability<br/>Humans<br/>Ku Autoantigen<br/>Mutation<br/>Nuclear Proteins--genetics<br/>Recombination, Genetic<br/>Recombinational DNA Repair<br/>Telomere--physiology<br/>Telomere Homeostasis<br/>Telomeric Repeat Binding Protein 2--metabolism