Nakka, Manjula <br/><br/>
Targeted disruption of the p160 coactivator interface of androgen receptor (AR) selectively inhibits AR activity in both androgen-dependent and castration-resistant AR-expressing prostate cancer cells.  [electronic resource] 

 -  The international journal of biochemistry & cell biology  Apr 2013 
 - 763-72 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1878-5875 
<br/><br/><label>Standard No.: </label>10.1016/j.biocel.2012.12.012  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Androgen Receptor Antagonists--chemistry<br/>Androgens--metabolism<br/>Animals<br/>Binding Sites--drug effects<br/>COS Cells<br/>Cell Line, Tumor<br/>Cell Proliferation--drug effects<br/>Chlorocebus aethiops<br/>Gene Expression Regulation, Neoplastic--drug effects<br/>Humans<br/>Male<br/>Molecular Targeted Therapy<br/>Nuclear Receptor Coactivators--chemistry<br/>Peptide Fragments--chemistry<br/>Prostate-Specific Antigen--antagonists & inhibitors<br/>Prostatic Neoplasms--pathology<br/>Protein Structure, Tertiary<br/>Receptors, Androgen--genetics<br/>Transcriptional Activation--drug effects