Gopinath, Sreelatha <br/><br/>
Cathepsin B and uPAR regulate self-renewal of glioma-initiating cells through GLI-regulated Sox2 and Bmi1 expression.  [electronic resource] 

 -  Carcinogenesis  Mar 2013 
 - 550-9 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1460-2180 
<br/><br/><label>Standard No.: </label>10.1093/carcin/bgs375  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>AC133 Antigen<br/>Animals<br/>Antigens, CD--metabolism<br/>Cathepsin B--genetics<br/>Cell Line, Tumor<br/>Cell Proliferation<br/>Cell Separation<br/>Female<br/>Flow Cytometry<br/>Gene Expression<br/>Gene Expression Regulation, Neoplastic<br/>Gene Knockdown Techniques<br/>Glioma--metabolism<br/>Glycoproteins--metabolism<br/>Hedgehog Proteins--metabolism<br/>Humans<br/>Mice<br/>Mice, Nude<br/>Neoplasm Transplantation<br/>Neoplastic Stem Cells--metabolism<br/>Peptides--metabolism<br/>Polycomb Repressive Complex 1--genetics<br/>RNA, Small Interfering--genetics<br/>Receptors, Urokinase Plasminogen Activator--genetics<br/>SOXB1 Transcription Factors--genetics<br/>Signal Transduction<br/>Transcription Factors--metabolism<br/>Transcriptional Activation<br/>Zinc Finger Protein GLI1