Inhibition of nephrin activation by c-mip through Csk-Cbp-Fyn axis plays a critical role in Angiotensin II-induced podocyte damage. [electronic resource]
- Cellular signalling Mar 2013
- 581-8 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1873-3913
10.1016/j.cellsig.2012.11.017 doi
Adaptor Proteins, Signal Transducing Angiotensin II--pharmacology Animals Carrier Proteins--antagonists & inhibitors Caspase 3--metabolism Cell Line Membrane Proteins--metabolism Mice Phosphoproteins--metabolism Phosphorylation Podocytes--drug effects Proto-Oncogene Proteins c-akt--metabolism Proto-Oncogene Proteins c-bcl-2--metabolism Proto-Oncogene Proteins c-fyn--metabolism RNA Interference RNA, Small Interfering--metabolism Signal Transduction Vasoconstrictor Agents--pharmacology bcl-Associated Death Protein--metabolism