Liu, Ai-Ran

Activation of canonical wnt pathway promotes differentiation of mouse bone marrow-derived MSCs into type II alveolar epithelial cells, confers resistance to oxidative stress, and promotes their migration to injured lung tissue in vitro. [electronic resource] - Journal of cellular physiology Jun 2013 - 1270-83 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1097-4652

10.1002/jcp.24282 doi


Acute Lung Injury--metabolism
Alveolar Epithelial Cells--drug effects
Animals
Biomarkers--metabolism
Bone Marrow Cells--drug effects
Cell Differentiation--drug effects
Cell Movement
Cell Proliferation
Cells, Cultured
Coculture Techniques
Culture Media, Conditioned--metabolism
Disease Models, Animal
Dose-Response Relationship, Drug
Glycogen Synthase Kinase 3--metabolism
Glycogen Synthase Kinase 3 beta
Hydrogen Peroxide--pharmacology
Lithium Chloride--pharmacology
Male
Mesenchymal Stem Cell Transplantation
Mesenchymal Stem Cells--drug effects
Mice
Mice, Inbred C57BL
Oxidants--pharmacology
Oxidative Stress--drug effects
Re-Epithelialization
Time Factors
Tissue Culture Techniques
Wnt Signaling Pathway--drug effects
Wnt3A Protein--metabolism
beta Catenin--metabolism