Travers, Mary E <br/><br/>
Insights into the molecular mechanism for type 2 diabetes susceptibility at the KCNQ1 locus from temporal changes in imprinting status in human islets.  [electronic resource] 

 -  Diabetes  Mar 2013 
 - 987-92 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1939-327X 
<br/><br/><label>Standard No.: </label>10.2337/db12-0819  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adult<br/>Alleles<br/>Chromosomes, Human, Pair 11--genetics<br/>Cohort Studies<br/>Cyclin-Dependent Kinase Inhibitor p57--genetics<br/>DNA Methylation<br/>Diabetes Mellitus, Type 2--genetics<br/>Fetal Development<br/>Gene Expression Regulation, Developmental<br/>Genetic Association Studies<br/>Genetic Loci<br/>Genetic Predisposition to Disease<br/>Humans<br/>Introns<br/>Islets of Langerhans--growth & development<br/>KCNQ1 Potassium Channel--genetics<br/>Pancreas--embryology<br/>Polymorphism, Single Nucleotide<br/>Potassium Channels, Voltage-Gated--genetics<br/>United Kingdom