Blohmke, Christoph J <br/><br/>
Atypical activation of the unfolded protein response in cystic fibrosis airway cells contributes to p38 MAPK-mediated innate immune responses.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  Dec 2012 
 - 5467-75 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1550-6606 
<br/><br/><label>Standard No.: </label>10.4049/jimmunol.1103661  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Cells, Cultured<br/>Cinnamates--pharmacology<br/>Cystic Fibrosis--complications<br/>Endoplasmic Reticulum--drug effects<br/>Endoplasmic Reticulum Stress--drug effects<br/>Epithelial Cells--immunology<br/>Eukaryotic Initiation Factor-2--genetics<br/>Flagellin--immunology<br/>Gene Expression Profiling<br/>Gene Expression Regulation--drug effects<br/>Humans<br/>Immunity, Innate--drug effects<br/>Interleukin-6--biosynthesis<br/>Lung--immunology<br/>Pneumonia--complications<br/>Pseudomonas aeruginosa--immunology<br/>Signal Transduction--drug effects<br/>Thiourea--analogs & derivatives<br/>Unfolded Protein Response--drug effects<br/>eIF-2 Kinase--genetics<br/>p38 Mitogen-Activated Protein Kinases--genetics