Burgess, Don E <br/><br/>
High-risk long QT syndrome mutations in the Kv7.1 (KCNQ1) pore disrupt the molecular basis for rapid K(+) permeation.  [electronic resource] 

 -  Biochemistry  Nov 2012 
 - 9076-85 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1520-4995 
<br/><br/><label>Standard No.: </label>10.1021/bi3009449  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adolescent<br/>Adult<br/>Amino Acid Sequence<br/>Child<br/>Female<br/>Humans<br/>KCNQ1 Potassium Channel--genetics<br/>Male<br/>Middle Aged<br/>Molecular Dynamics Simulation<br/>Mutation, Missense<br/>Potassium--metabolism<br/>Potassium Channels, Voltage-Gated--genetics<br/>Risk<br/>Romano-Ward Syndrome--genetics<br/>Sequence Alignment