Huang, Yi-Hsien <br/><br/>
STAT1 activation by venous malformations mutant Tie2-R849W antagonizes VEGF-A-mediated angiogenic response partly via reduced bFGF production.  [electronic resource] 

 -  Angiogenesis  Jan 2013 
 - 207-22 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1573-7209 
<br/><br/><label>Standard No.: </label>10.1007/s10456-012-9313-x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Base Sequence<br/>Cell Movement--drug effects<br/>Cell Nucleus--drug effects<br/>Endothelial Cells--metabolism<br/>Fibroblast Growth Factor 2--biosynthesis<br/>Human Umbilical Vein Endothelial Cells--drug effects<br/>Humans<br/>Molecular Sequence Data<br/>Mutation, Missense--genetics<br/>Myocytes, Smooth Muscle--drug effects<br/>Neovascularization, Physiologic--drug effects<br/>Neutralization Tests<br/>Phosphorylation--drug effects<br/>Phosphotyrosine--metabolism<br/>Promoter Regions, Genetic--genetics<br/>Protein Binding--drug effects<br/>Protein Transport--drug effects<br/>Receptor, TIE-2--genetics<br/>STAT1 Transcription Factor--metabolism<br/>STAT3 Transcription Factor--metabolism<br/>Vascular Endothelial Growth Factor A--pharmacology<br/>Vascular Malformations--metabolism