Guzmán-Ramos, Kioko <br/><br/>
Restoration of dopamine release deficits during object recognition memory acquisition attenuates cognitive impairment in a triple transgenic mice model of Alzheimer's disease.  [electronic resource] 

 -  Learning & memory (Cold Spring Harbor, N.Y.)  Sep 2012 
 - 453-60 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1549-5485 
<br/><br/><label>Standard No.: </label>10.1101/lm.026070.112  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Age Factors<br/>Alzheimer Disease--complications<br/>Amyloid beta-Peptides--metabolism<br/>Amyloid beta-Protein Precursor--genetics<br/>Animals<br/>Brain--drug effects<br/>Cognition Disorders--drug therapy<br/>Disease Models, Animal<br/>Dopamine--metabolism<br/>Dopamine Uptake Inhibitors--therapeutic use<br/>Exploratory Behavior--drug effects<br/>Glutamic Acid--metabolism<br/>Humans<br/>Male<br/>Memory Disorders--drug therapy<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Transgenic<br/>Microdialysis<br/>Mutation--genetics<br/>Nomifensine--therapeutic use<br/>Norepinephrine--metabolism<br/>Photic Stimulation<br/>Presenilin-1--genetics<br/>Recognition, Psychology--drug effects<br/>Statistics, Nonparametric<br/>tau Proteins--genetics