Zhao, Ting C <br/><br/>
Disruption of Nox2 and TNFRp55/p75 eliminates cardioprotection induced by anisomycin.  [electronic resource] 

 -  American journal of physiology. Heart and circulatory physiology  Nov 2012 
 - H1263-72 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1522-1539 
<br/><br/><label>Standard No.: </label>10.1152/ajpheart.00306.2012  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Anisomycin--administration & dosage<br/>Disease Models, Animal<br/>Enzyme Activation<br/>Enzyme Activators--administration & dosage<br/>Injections, Intraperitoneal<br/>Male<br/>Membrane Glycoproteins--deficiency<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Myocardial Infarction--drug therapy<br/>Myocardial Reperfusion Injury--drug therapy<br/>Myocardium--metabolism<br/>NADPH Oxidase 2<br/>NADPH Oxidases--deficiency<br/>Protein Kinase Inhibitors--pharmacology<br/>Receptors, Tumor Necrosis Factor, Type I--deficiency<br/>Receptors, Tumor Necrosis Factor, Type II--deficiency<br/>Signal Transduction--drug effects<br/>Superoxides--metabolism<br/>Time Factors<br/>Tumor Necrosis Factor-alpha--metabolism<br/>Ventricular Function, Left--drug effects<br/>p38 Mitogen-Activated Protein Kinases--antagonists & inhibitors