El Malki, Khalifa <br/><br/>
An alternative pathway of imiquimod-induced psoriasis-like skin inflammation in the absence of interleukin-17 receptor a signaling.  [electronic resource] 

 -  The Journal of investigative dermatology  Feb 2013 
 - 441-51 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1523-1747 
<br/><br/><label>Standard No.: </label>10.1038/jid.2012.318  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adjuvants, Immunologic--pharmacology<br/>Aminoquinolines--pharmacology<br/>Animals<br/>Disease Models, Animal<br/>Female<br/>Imiquimod<br/>Interleukin-17--immunology<br/>Interleukin-6--immunology<br/>Interleukins--immunology<br/>Macrophages--drug effects<br/>Mice<br/>Mice, Knockout<br/>Neutrophil Infiltration--drug effects<br/>Psoriasis--chemically induced<br/>Receptors, Interleukin-17--genetics<br/>Signal Transduction--drug effects<br/>Skin--immunology<br/>Interleukin-22