Hamamoto, S <br/><br/>
Vildagliptin preserves the mass and function of pancreatic β cells via the developmental regulation and suppression of oxidative and endoplasmic reticulum stress in a mouse model of diabetes.  [electronic resource] 

 -  Diabetes, obesity & metabolism  Feb 2013 
 - 153-63 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1463-1326 
<br/><br/><label>Standard No.: </label>10.1111/dom.12005  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adamantane--analogs & derivatives<br/>Animals<br/>Apoptosis<br/>Blood Glucose--metabolism<br/>Cell Proliferation<br/>Diabetes Mellitus, Experimental--drug therapy<br/>Endoplasmic Reticulum Stress--drug effects<br/>Gene Expression Regulation, Developmental<br/>Immunohistochemistry<br/>Insulin--metabolism<br/>Insulin-Secreting Cells--metabolism<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Nitriles--pharmacology<br/>Oxidative Stress--drug effects<br/>Pyrrolidines--pharmacology<br/>Real-Time Polymerase Chain Reaction<br/>Triglycerides--metabolism<br/>Up-Regulation<br/>Vildagliptin