Winkler, Ingrid G <br/><br/>
B-lymphopoiesis is stopped by mobilizing doses of G-CSF and is rescued by overexpression of the anti-apoptotic protein Bcl2.  [electronic resource] 

 -  Haematologica  Mar 2013 
 - 325-33 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1592-8721 
<br/><br/><label>Standard No.: </label>10.3324/haematol.2012.069260  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>B-Lymphocyte Subsets--drug effects<br/>Benzylamines<br/>Bone Marrow--drug effects<br/>Cyclams<br/>Cyclophosphamide--pharmacology<br/>Gene Expression<br/>Granulocyte Colony-Stimulating Factor--administration & dosage<br/>Hematopoietic Stem Cell Mobilization<br/>Heterocyclic Compounds--pharmacology<br/>Lymphopoiesis--drug effects<br/>Male<br/>Mice<br/>Precursor Cells, B-Lymphoid--drug effects<br/>Proto-Oncogene Proteins c-bcl-2--genetics<br/>Spleen--drug effects<br/>Tumor Necrosis Factor-alpha--antagonists & inhibitors