Shields, Shannon D
Sodium channel Na(v)1.7 is essential for lowering heat pain threshold after burn injury. [electronic resource]
- The Journal of neuroscience : the official journal of the Society for Neuroscience Aug 2012
- 10819-32 p. digital
Publication Type: Journal Article; Research Support, U.S. Gov't, Non-P.H.S.
ISSN: 1529-2401
Standard No.: 10.1523/JNEUROSCI.0304-12.2012 doi
Subjects--Topical Terms: Activating Transcription Factor 3--metabolism Analysis of Variance Animals Biophysics Burns--complications Calcitonin Gene-Related Peptide--metabolism Calcium--metabolism Cells, Cultured Cytokines--metabolism Disease Models, Animal Edema--etiology Electric Stimulation Functional Laterality Ganglia, Spinal--cytology Glycoproteins--metabolism Hot Temperature--adverse effects Hyperalgesia--etiology Membrane Potentials--drug effects Mice Mice, Inbred C57BL Mice, Transgenic NAV1.7 Voltage-Gated Sodium Channel NAV1.8 Voltage-Gated Sodium Channel NAV1.9 Voltage-Gated Sodium Channel Neuralgia--etiology Neuroglia--pathology Pain Threshold--physiology Patch-Clamp Techniques Proteins--genetics RNA, Messenger--metabolism RNA, Untranslated Sensory Receptor Cells--drug effects Sodium Channel Blockers--pharmacology Sodium Channels--deficiency Tetrodotoxin--pharmacology Transfection