Shields, Shannon D

Sodium channel Na(v)1.7 is essential for lowering heat pain threshold after burn injury. [electronic resource] - The Journal of neuroscience : the official journal of the Society for Neuroscience Aug 2012 - 10819-32 p. digital

Publication Type: Journal Article; Research Support, U.S. Gov't, Non-P.H.S.

1529-2401

10.1523/JNEUROSCI.0304-12.2012 doi


Activating Transcription Factor 3--metabolism
Analysis of Variance
Animals
Biophysics
Burns--complications
Calcitonin Gene-Related Peptide--metabolism
Calcium--metabolism
Cells, Cultured
Cytokines--metabolism
Disease Models, Animal
Edema--etiology
Electric Stimulation
Functional Laterality
Ganglia, Spinal--cytology
Glycoproteins--metabolism
Hot Temperature--adverse effects
Hyperalgesia--etiology
Membrane Potentials--drug effects
Mice
Mice, Inbred C57BL
Mice, Transgenic
NAV1.7 Voltage-Gated Sodium Channel
NAV1.8 Voltage-Gated Sodium Channel
NAV1.9 Voltage-Gated Sodium Channel
Neuralgia--etiology
Neuroglia--pathology
Pain Threshold--physiology
Patch-Clamp Techniques
Proteins--genetics
RNA, Messenger--metabolism
RNA, Untranslated
Sensory Receptor Cells--drug effects
Sodium Channel Blockers--pharmacology
Sodium Channels--deficiency
Tetrodotoxin--pharmacology
Transfection