Vilahur, Gemma <br/><br/>
Reperfusion-triggered stress protein response in the myocardium is blocked by post-conditioning. Systems biology pathway analysis highlights the key role of the canonical aryl-hydrocarbon receptor pathway.  [electronic resource] 

 -  European heart journal  Jul 2013 
 - 2082-93 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1522-9645 
<br/><br/><label>Standard No.: </label>10.1093/eurheartj/ehs211  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--physiology<br/>Aryl Hydrocarbon Receptor Nuclear Translocator--metabolism<br/>Caspase 8--metabolism<br/>Coronary Vessels--pathology<br/>Deoxyuracil Nucleotides--metabolism<br/>Echocardiography<br/>Heat-Shock Proteins--physiology<br/>Ischemic Postconditioning--methods<br/>Myocardial Contraction--physiology<br/>Myocardial Reperfusion Injury--metabolism<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Proteome--metabolism<br/>Proteomics<br/>Proto-Oncogene Proteins c-bcl-2--metabolism<br/>Random Allocation<br/>Receptors, Aryl Hydrocarbon--metabolism<br/>Signal Transduction--physiology<br/>Sus scrofa<br/>Systems Biology<br/>TOR Serine-Threonine Kinases--metabolism