Simão, Fabrício <br/><br/>
Resveratrol prevents CA1 neurons against ischemic injury by parallel modulation of both GSK-3β and CREB through PI3-K/Akt pathways.  [electronic resource] 

 -  The European journal of neuroscience  Oct 2012 
 - 2899-905 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1460-9568 
<br/><br/><label>Standard No.: </label>10.1111/j.1460-9568.2012.08229.x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--drug effects<br/>Brain Ischemia--drug therapy<br/>CA1 Region, Hippocampal--drug effects<br/>Chromones--pharmacology<br/>Cyclic AMP Response Element-Binding Protein--metabolism<br/>Glycogen Synthase Kinase 3--metabolism<br/>Glycogen Synthase Kinase 3 beta<br/>Male<br/>Morpholines--pharmacology<br/>Neurons--drug effects<br/>Neuroprotective Agents--pharmacology<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Phosphoinositide-3 Kinase Inhibitors<br/>Phosphorylation<br/>Proto-Oncogene Proteins c-akt--metabolism<br/>Rats<br/>Rats, Wistar<br/>Reperfusion Injury--drug therapy<br/>Resveratrol<br/>Signal Transduction--drug effects<br/>Stilbenes--pharmacology