Vissers, Marloes <br/><br/>
Respiratory syncytial virus infection augments NOD2 signaling in an IFN-β-dependent manner in human primary cells.  [electronic resource] 

 -  European journal of immunology  Oct 2012 
 - 2727-35 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1521-4141 
<br/><br/><label>Standard No.: </label>10.1002/eji.201242396  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acetylmuramyl-Alanyl-Isoglutamine--immunology<br/>Cells, Cultured<br/>Crohn Disease--immunology<br/>DEAD Box Protein 58<br/>DEAD-box RNA Helicases--genetics<br/>Humans<br/>Infant<br/>Infant, Newborn<br/>Inflammation Mediators--metabolism<br/>Interferon-beta--metabolism<br/>Mutation--genetics<br/>Nod2 Signaling Adaptor Protein--genetics<br/>Primary Cell Culture<br/>RNA, Viral--metabolism<br/>Receptors, Immunologic<br/>Respiratory Syncytial Virus Infections--immunology<br/>Respiratory Syncytial Viruses--physiology<br/>Signal Transduction<br/>Toll-Like Receptor 3--genetics<br/>Up-Regulation