Siddiqui, Almas <br/><br/>
Selective binding of nuclear alpha-synuclein to the PGC1alpha promoter under conditions of oxidative stress may contribute to losses in mitochondrial function: implications for Parkinson's disease.  [electronic resource] 

 -  Free radical biology & medicine  Aug 2012 
 - 993-1003 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1873-4596 
<br/><br/><label>Standard No.: </label>10.1016/j.freeradbiomed.2012.05.024  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aged<br/>Animals<br/>Cell Nucleus--metabolism<br/>Chromatin--metabolism<br/>Chromatin Immunoprecipitation<br/>Electron Transport Complex I--metabolism<br/>Heat-Shock Proteins--genetics<br/>Humans<br/>Mice<br/>Mice, Transgenic<br/>Middle Aged<br/>Mitochondria--metabolism<br/>Oxidative Stress<br/>PC12 Cells<br/>Parkinson Disease--metabolism<br/>Peroxisome Proliferator-Activated Receptor Gamma Coactivator 1-alpha<br/>Promoter Regions, Genetic<br/>Protein Binding<br/>Rats<br/>Transcription Factors--genetics<br/>alpha-Synuclein--metabolism