Foley, Andrew G <br/><br/>
Class I histone deacetylase inhibition ameliorates social cognition and cell adhesion molecule plasticity deficits in a rodent model of autism spectrum disorder.  [electronic resource] 

 -  Neuropharmacology  Sep 2012 
 - 750-60 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1873-7064 
<br/><br/><label>Standard No.: </label>10.1016/j.neuropharm.2012.05.042  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Behavior, Animal--drug effects<br/>Child<br/>Child Development Disorders, Pervasive--drug therapy<br/>Cognition Disorders--etiology<br/>Dentate Gyrus--drug effects<br/>Disease Models, Animal<br/>Female<br/>Histone Deacetylase 1--antagonists & inhibitors<br/>Histone Deacetylase Inhibitors--therapeutic use<br/>Humans<br/>Male<br/>Molecular Targeted Therapy<br/>Nerve Tissue Proteins--metabolism<br/>Neural Cell Adhesion Molecule L1--metabolism<br/>Neuronal Plasticity--drug effects<br/>Neurons--drug effects<br/>Pregnancy<br/>Prenatal Exposure Delayed Effects<br/>Rats<br/>Rats, Wistar<br/>Sialic Acids--metabolism<br/>Social Behavior