Li, Xiang-dong <br/><br/>
Phosphorylation of endothelial NOS contributes to simvastatin protection against myocardial no-reflow and infarction in reperfused swine hearts: partially via the PKA signaling pathway.  [electronic resource] 

 -  Acta pharmacologica Sinica  Jul 2012 
 - 879-87 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1745-7254 
<br/><br/><label>Standard No.: </label>10.1038/aps.2012.27  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Anticholesteremic Agents--therapeutic use<br/>Creatine Kinase--blood<br/>Cyclic AMP Response Element-Binding Protein--metabolism<br/>Cyclic AMP-Dependent Protein Kinases--metabolism<br/>Heart--drug effects<br/>Hemodynamics--drug effects<br/>Myocardial Infarction--enzymology<br/>Myocardial Reperfusion Injury--enzymology<br/>Myocardium--enzymology<br/>Nitric Oxide Synthase Type III--metabolism<br/>Phosphorylation--drug effects<br/>Signal Transduction--drug effects<br/>Simvastatin--therapeutic use<br/>Swine<br/>Swine, Miniature