Conrad, Kelly L

Yohimbine depresses excitatory transmission in BNST and impairs extinction of cocaine place preference through orexin-dependent, norepinephrine-independent processes. [electronic resource] - Neuropsychopharmacology : official publication of the American College of Neuropsychopharmacology Sep 2012 - 2253-66 p. digital

Publication Type: Journal Article; Research Support, N.I.H., Extramural

1740-634X

10.1038/npp.2012.76 doi


Adrenergic alpha-1 Receptor Antagonists--pharmacology
Adrenergic alpha-2 Receptor Antagonists--pharmacology
Animals
Behavior, Animal--drug effects
Cocaine--pharmacology
Drug-Seeking Behavior--drug effects
Extinction, Psychological--drug effects
Intracellular Signaling Peptides and Proteins--pharmacology
Male
Mice
Mice, Inbred C57BL
Mice, Knockout
Morpholines--pharmacology
Neuropeptides--pharmacology
Neurotransmitter Agents--pharmacology
Norepinephrine--physiology
Norepinephrine Plasma Membrane Transport Proteins--antagonists & inhibitors
Orexin Receptors
Orexins
Patch-Clamp Techniques
Reboxetine
Receptors, Adrenergic, alpha-2--genetics
Receptors, Corticotropin-Releasing Hormone--antagonists & inhibitors
Receptors, Neuropeptide--antagonists & inhibitors
Reward
Septal Nuclei--drug effects
Synaptic Transmission--drug effects
Yohimbine--pharmacology
CRF Receptor, Type 1