Tang, J <br/><br/>
CD147 induces UPR to inhibit apoptosis and chemosensitivity by increasing the transcription of Bip in hepatocellular carcinoma.  [electronic resource] 

 -  Cell death and differentiation  Nov 2012 
 - 1779-90 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1476-5403 
<br/><br/><label>Standard No.: </label>10.1038/cdd.2012.60  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Antineoplastic Agents--pharmacology<br/>Apoptosis--drug effects<br/>Basigin--chemistry<br/>Biomarkers, Tumor--metabolism<br/>Carcinoma, Hepatocellular--drug therapy<br/>Cell Line, Tumor<br/>Doxorubicin--pharmacology<br/>Endoplasmic Reticulum Chaperone BiP<br/>Endoplasmic Reticulum Stress<br/>Focal Adhesion Kinase 1--metabolism<br/>HEK293 Cells<br/>Heat-Shock Proteins--genetics<br/>Hep G2 Cells<br/>Humans<br/>Liver Neoplasms--drug therapy<br/>Mice<br/>Mice, Nude<br/>Promoter Regions, Genetic<br/>RNA Interference<br/>RNA, Small Interfering--metabolism<br/>Transcription Factors, TFII--metabolism<br/>Transcription, Genetic<br/>Unfolded Protein Response<br/>src-Family Kinases--metabolism