Häcker, Hans <br/><br/>
NIK prevents the development of hypereosinophilic syndrome-like disease in mice independent of IKKα activation.  [electronic resource] 

 -  Journal of immunology (Baltimore, Md. : 1950)  May 2012 
 - 4602-10 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1550-6606 
<br/><br/><label>Standard No.: </label>10.4049/jimmunol.1200021  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Enzyme Activation--genetics<br/>Hypereosinophilic Syndrome--genetics<br/>I-kappa B Kinase--genetics<br/>Inflammation--genetics<br/>Mice<br/>Mice, Knockout<br/>Mutation<br/>Protein Serine-Threonine Kinases--genetics<br/>Protein Structure, Secondary<br/>Th2 Cells--immunology<br/>NF-kappaB-Inducing Kinase