Gerfaux, J <br/><br/>
A common cellular pathway for v-mos and v-Ki-ras is not required for v-Ki-ras-induced tumorigenicity in a nonmalignant, v-mos-expressing revertant cell.  [electronic resource] 

 -  Molecular carcinogenesis  1990 
 - 103-13 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0899-1987 
<br/><br/><label>Standard No.: </label>10.1002/mc.2940030208  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cell Transformation, Viral<br/>Chimera<br/>Collagen--biosynthesis<br/>Down-Regulation<br/>Mice<br/>Mice, Inbred BALB C<br/>Mice, Nude<br/>Moloney murine sarcoma virus--genetics<br/>Oncogene Proteins v-mos<br/>Oncogenes<br/>Promoter Regions, Genetic<br/>Protein-Tyrosine Kinases--genetics<br/>Proto-Oncogene Proteins--genetics<br/>Proto-Oncogene Proteins p21(ras)<br/>Retroviridae Proteins, Oncogenic--genetics<br/>Sarcoma Viruses, Murine--genetics<br/>Tumor Cells, Cultured