Taube, Janis M <br/><br/>
Colocalization of inflammatory response with B7-h1 expression in human melanocytic lesions supports an adaptive resistance mechanism of immune escape.  [electronic resource] 

 -  Science translational medicine  Mar 2012 
 - 127ra37 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1946-6242 
<br/><br/><label>Standard No.: </label>10.1126/scitranslmed.3003689  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adaptive Immunity--immunology<br/>Adolescent<br/>Adult<br/>Aged<br/>Aged, 80 and over<br/>B7-H1 Antigen--genetics<br/>Child<br/>Demography<br/>Female<br/>Follow-Up Studies<br/>Gene Expression Regulation, Neoplastic<br/>Humans<br/>Inflammation--complications<br/>Interferon-gamma--immunology<br/>Laser Capture Microdissection<br/>Lymphocytes, Tumor-Infiltrating--immunology<br/>Male<br/>Melanocytes--immunology<br/>Melanoma--complications<br/>Middle Aged<br/>Neoplasm Staging<br/>Reverse Transcriptase Polymerase Chain Reaction<br/>Skin Neoplasms--complications<br/>Survival Analysis<br/>Tumor Escape--immunology<br/>Young Adult