Simar, David <br/><br/>
Heat shock proteins induction reduces stress kinases activation, potentially improving insulin signalling in monocytes from obese subjects.  [electronic resource] 

 -  Cell stress & chaperones  Sep 2012 
 - 615-21 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1466-1268 
<br/><br/><label>Standard No.: </label>10.1007/s12192-012-0336-4  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adult<br/>Female<br/>Glucose--metabolism<br/>Glucose Tolerance Test<br/>Glucose Transporter Type 4--metabolism<br/>HSP27 Heat-Shock Proteins--metabolism<br/>HSP72 Heat-Shock Proteins--metabolism<br/>Heat-Shock Proteins--metabolism<br/>Humans<br/>I-kappa B Kinase--metabolism<br/>Insulin--metabolism<br/>Insulin Receptor Substrate Proteins--metabolism<br/>JNK Mitogen-Activated Protein Kinases--metabolism<br/>Male<br/>Middle Aged<br/>Monocytes--metabolism<br/>Obesity--metabolism<br/>Signal Transduction