Wigan, Matthew <br/><br/>
A UVR-induced G2-phase checkpoint response to ssDNA gaps produced by replication fork bypass of unrepaired lesions is defective in melanoma.  [electronic resource] 

 -  The Journal of investigative dermatology  Jun 2012 
 - 1681-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1523-1747 
<br/><br/><label>Standard No.: </label>10.1038/jid.2012.41  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>CDC2 Protein Kinase--metabolism<br/>Cell Cycle Checkpoints--genetics<br/>Cell Line, Tumor<br/>Cyclin B1--metabolism<br/>DNA Replication--genetics<br/>DNA, Single-Stranded--genetics<br/>DNA-Binding Proteins--physiology<br/>Epidermal Cells<br/>Epidermis--radiation effects<br/>G1 Phase--genetics<br/>G2 Phase--genetics<br/>Humans<br/>Melanoma--genetics<br/>Mutation--genetics<br/>S Phase--genetics<br/>Skin Neoplasms--genetics<br/>Ultraviolet Rays--adverse effects