BMPR-II deficiency elicits pro-proliferative and anti-apoptotic responses through the activation of TGFβ-TAK1-MAPK pathways in PAH. [electronic resource]
- Human molecular genetics Jun 2012
- 2548-58 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1460-2083
10.1093/hmg/dds073 doi
Animals Apoptosis Bone Morphogenetic Protein Receptors, Type II--metabolism Cell Proliferation Cells, Cultured Familial Primary Pulmonary Hypertension Hypertension, Pulmonary--metabolism MAP Kinase Kinase Kinases--metabolism Mice Pulmonary Artery--metabolism Rats Signal Transduction Transforming Growth Factor beta--metabolism