Ptasinska, A <br/><br/>
Depletion of RUNX1/ETO in t(8;21) AML cells leads to genome-wide changes in chromatin structure and transcription factor binding.  [electronic resource] 

 -  Leukemia  Aug 2012 
 - 1829-41 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1476-5551 
<br/><br/><label>Standard No.: </label>10.1038/leu.2012.49  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acetylation<br/>Binding Sites<br/>CCAAT-Binding Factor--genetics<br/>Cell Differentiation--genetics<br/>Cell Line, Tumor<br/>Chromatin--genetics<br/>Chromosomes, Human, Pair 21<br/>Chromosomes, Human, Pair 8<br/>Cluster Analysis<br/>Core Binding Factor Alpha 2 Subunit--genetics<br/>Gene Expression Profiling<br/>Gene Silencing<br/>Histones--metabolism<br/>Humans<br/>Leukemia, Myeloid, Acute--genetics<br/>Mutation<br/>Oncogene Proteins, Fusion--genetics<br/>Protein Binding<br/>Proto-Oncogene Proteins--genetics<br/>RNA Polymerase II--metabolism<br/>RUNX1 Translocation Partner 1 Protein<br/>Transcription Factors--genetics<br/>Transcriptional Activation<br/>Translocation, Genetic