Oomizu, Souichi <br/><br/>
Galectin-9 suppresses Th17 cell development in an IL-2-dependent but Tim-3-independent manner.  [electronic resource] 

 -  Clinical immunology (Orlando, Fla.)  Apr 2012 
 - 51-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1521-7035 
<br/><br/><label>Standard No.: </label>10.1016/j.clim.2012.01.004  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Acetylgalactosamine--analogs & derivatives<br/>Animals<br/>Benzyl Compounds--pharmacology<br/>Cells, Cultured<br/>Encephalomyelitis, Autoimmune, Experimental--genetics<br/>Female<br/>Flow Cytometry<br/>Forkhead Transcription Factors--immunology<br/>Galectins--genetics<br/>Hepatitis A Virus Cellular Receptor 2<br/>Interleukin-17--immunology<br/>Interleukin-2--immunology<br/>Interleukin-2 Receptor alpha Subunit--immunology<br/>Interleukin-6--pharmacology<br/>Male<br/>Mice<br/>Mice, Inbred C57BL<br/>Mice, Knockout<br/>Receptors, Virus--immunology<br/>T-Lymphocytes, Regulatory--immunology<br/>Th17 Cells--drug effects<br/>Transforming Growth Factor beta1--pharmacology