Mitchell, Callum <br/><br/>
A Ca²⁺-dependent chloride current and Ca²⁺ influx via Ca(v)1.2 ion channels play major roles in P2Y receptor-mediated pulmonary vasoconstriction.  [electronic resource] 

 -  British journal of pharmacology  Jun 2012 
 - 1503-12 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1476-5381 
<br/><br/><label>Standard No.: </label>10.1111/j.1476-5381.2012.01892.x  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Adenosine Triphosphate--metabolism<br/>Animals<br/>Calcium Channels, L-Type--metabolism<br/>Calcium Signaling--drug effects<br/>Chloride Channels--antagonists & inhibitors<br/>Endothelium, Vascular--physiology<br/>In Vitro Techniques<br/>Kinetics<br/>Male<br/>Membrane Potentials--drug effects<br/>Membrane Transport Modulators--pharmacology<br/>Myography<br/>Pulmonary Artery--drug effects<br/>Purinergic Antagonists--pharmacology<br/>Rats<br/>Rats, Sprague-Dawley<br/>Receptors, Purinergic P2X1--metabolism<br/>Receptors, Purinergic P2Y--metabolism<br/>Uridine Diphosphate--metabolism<br/>Uridine Triphosphate--metabolism<br/>Vasoconstriction--drug effects