Kashihara, Toshihide

Chronic receptor-mediated activation of Gi/o proteins alters basal t-tubular and sarcolemmal L-type Ca²⁺ channel activity through phosphatases in heart failure. [electronic resource] - American journal of physiology. Heart and circulatory physiology Apr 2012 - H1645-54 p. digital

Publication Type: Journal Article; Research Support, Non-U.S. Gov't

1522-1539

10.1152/ajpheart.00589.2011 doi


3-Pyridinecarboxylic acid, 1,4-dihydro-2,6-dimethyl-5-nitro-4-(2-(trifluoromethyl)phenyl)-, Methyl ester--pharmacology
Adrenergic beta-Agonists--pharmacology
Algorithms
Animals
Blood Pressure--drug effects
Calcium Channel Agonists--pharmacology
Calcium Channels, L-Type--drug effects
Cyclic AMP-Dependent Protein Kinases--metabolism
GTP-Binding Protein alpha Subunits, Gi-Go--drug effects
Heart Failure--diagnostic imaging
Heart Rate--drug effects
Isoproterenol--pharmacology
Male
Membrane Potentials--drug effects
Mice
Mice, Inbred C57BL
Microtubules--drug effects
Myocardium--pathology
Myocytes, Cardiac--physiology
Patch-Clamp Techniques
Pertussis Toxin--pharmacology
Phosphoprotein Phosphatases--antagonists & inhibitors
Receptors, G-Protein-Coupled--drug effects
Sarcolemma--drug effects
Ultrasonography