Electroacupuncture pretreatment attenuates cerebral ischemic injury through α7 nicotinic acetylcholine receptor-mediated inhibition of high-mobility group box 1 release in rats. [electronic resource]
- Journal of neuroinflammation Jan 2012
- 24 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1742-2094
10.1186/1742-2094-9-24 doi
Animals Brain Injuries--prevention & control Bridged Bicyclo Compounds, Heterocyclic--therapeutic use Bungarotoxins--pharmacology Cells, Cultured Cerebral Cortex--cytology Cerebral Infarction--etiology Disease Models, Animal Electroacupuncture--methods Embryo, Mammalian Gene Expression Regulation--drug effects Glucose--deficiency HMGB1 Protein--metabolism Hypoxia--therapy In Situ Nick-End Labeling Infarction, Middle Cerebral Artery--complications Injections, Intraventricular L-Lactate Dehydrogenase--metabolism Male Neurons--drug effects Neuroprotective Agents--therapeutic use Phosphopyruvate Hydratase--metabolism Protein Binding--drug effects Quinuclidines--therapeutic use Rats Rats, Sprague-Dawley Receptors, Nicotinic--metabolism Statistics, Nonparametric Tetrazolium Salts Thiazoles alpha7 Nicotinic Acetylcholine Receptor