Unbalance of CB1 receptors expressed in GABAergic and glutamatergic neurons in a transgenic mouse model of Huntington's disease. [electronic resource]
- Neurobiology of disease Mar 2012
- 983-91 p. digital
Publication Type: Journal Article
1095-953X
10.1016/j.nbd.2011.12.017 doi
Action Potentials--drug effects Analysis of Variance Animals Benzoxazines--pharmacology Brain--pathology Disease Models, Animal Dronabinol--analogs & derivatives Drug Interactions Electric Stimulation Excitatory Amino Acid Agents--pharmacology Glutamates--metabolism Humans Huntingtin Protein Huntington Disease--pathology In Vitro Techniques Mice Mice, Transgenic Microscopy, Immunoelectron Morpholines--pharmacology Motor Activity--drug effects Naphthalenes--pharmacology Nerve Tissue Proteins--genetics Neurons--drug effects Nuclear Proteins--genetics Patch-Clamp Techniques Piperidines--pharmacology Potassium--pharmacology Pyrazoles--pharmacology Receptor, Cannabinoid, CB1--agonists Statistics, Nonparametric Synaptosomes--drug effects Trinucleotide Repeats--genetics Tritium--metabolism Vesicular Glutamate Transport Protein 1--metabolism Vesicular Inhibitory Amino Acid Transport Proteins--metabolism gamma-Aminobutyric Acid--metabolism