Poling, Matthew C <br/><br/>
Sexually dimorphic testosterone secretion in prenatal and neonatal mice is independent of kisspeptin-Kiss1r and GnRH signaling.  [electronic resource] 

 -  Endocrinology  Feb 2012 
 - 782-93 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1945-7170 
<br/><br/><label>Standard No.: </label>10.1210/en.2011-1838  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Animals, Newborn--metabolism<br/>Female<br/>Fetus--metabolism<br/>Gene Expression Regulation, Developmental--physiology<br/>Gonadotropin-Releasing Hormone--genetics<br/>Kisspeptins--genetics<br/>Male<br/>Mice<br/>Mice, Knockout<br/>Neurokinin B--genetics<br/>Receptors, G-Protein-Coupled--genetics<br/>Receptors, Kisspeptin-1<br/>Sex Characteristics<br/>Signal Transduction<br/>Testis--growth & development<br/>Testosterone--metabolism