Sauerbeck, Andrew <br/><br/>
Traumatic brain injury and trichloroethylene exposure interact and produce functional, histological, and mitochondrial deficits.  [electronic resource] 

 -  Experimental neurology  Mar 2012 
 - 85-94 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1090-2430 
<br/><br/><label>Standard No.: </label>10.1016/j.expneurol.2011.12.012  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Brain Injuries--complications<br/>Corpus Striatum--pathology<br/>Disease Models, Animal<br/>Dopamine Plasma Membrane Transport Proteins--metabolism<br/>Gene Expression Regulation--drug effects<br/>Male<br/>Mitochondrial Diseases--etiology<br/>Motor Activity--drug effects<br/>Movement Disorders--etiology<br/>Oxygen Consumption--drug effects<br/>Rats<br/>Rats, Inbred F344<br/>Receptors, Dopamine D2--metabolism<br/>Rotarod Performance Test<br/>Solvents--toxicity<br/>Trichloroethylene--toxicity<br/>Tyrosine 3-Monooxygenase--metabolism