Aso, Ester
Amyloid generation and dysfunctional immunoproteasome activation with disease progression in animal model of familial Alzheimer's disease. [electronic resource]
- Brain pathology (Zurich, Switzerland) Sep 2012
- 636-53 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
ISSN: 1750-3639
Standard No.: 10.1111/j.1750-3639.2011.00560.x doi
Subjects--Topical Terms: Age Factors Alzheimer Disease--complications Amyloid beta-Peptides--metabolism Amyloid beta-Protein Precursor--genetics Animals Aspartic Acid Endopeptidases--genetics Avoidance Learning--physiology Brain--metabolism Cognition Disorders--etiology Cysteine Endopeptidases--metabolism Disease Models, Animal Disease Progression Gene Expression Regulation--genetics Humans Mice Mice, Inbred C57BL Mice, Transgenic Microscopy, Electron, Transmission Microscopy, Immunoelectron Mitogen-Activated Protein Kinases Mutation--genetics Neuropsychological Tests Plaque, Amyloid--metabolism Presenilin-1--genetics Proteasome Endopeptidase Complex--genetics RNA, Messenger--metabolism Recognition, Psychology--physiology Signal Transduction--genetics Superoxide Dismutase--metabolism Superoxide Dismutase-1 Ubiquitin Thiolesterase--metabolism alpha-Synuclein--metabolism tau Proteins--metabolism