Wang, Li-E <br/><br/>
Roles of genetic variants in the PI3K and RAS/RAF pathways in susceptibility to endometrial cancer and clinical outcomes.  [electronic resource] 

 -  Journal of cancer research and clinical oncology  Mar 2012 
 - 377-85 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1432-1335 
<br/><br/><label>Standard No.: </label>10.1007/s00432-011-1103-0  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Aged<br/>Case-Control Studies<br/>Class I Phosphatidylinositol 3-Kinases<br/>Endometrial Neoplasms--genetics<br/>Female<br/>Genetic Predisposition to Disease<br/>Genotype<br/>Humans<br/>Kaplan-Meier Estimate<br/>Logistic Models<br/>Middle Aged<br/>Odds Ratio<br/>PTEN Phosphohydrolase--genetics<br/>Phosphatidylinositol 3-Kinases--genetics<br/>Pilot Projects<br/>Polymorphism, Single Nucleotide<br/>Proportional Hazards Models<br/>Proto-Oncogene Proteins c-akt--genetics<br/>Proto-Oncogene Proteins p21(ras)--genetics<br/>Risk Assessment<br/>Risk Factors<br/>Signal Transduction--genetics<br/>TOR Serine-Threonine Kinases--genetics<br/>Treatment Outcome<br/>raf Kinases--genetics