Su, Wen-Hong <br/><br/>
Radiation-induced increase in cell migration and metastatic potential of cervical cancer cells operates via the K-Ras pathway.  [electronic resource] 

 -  The American journal of pathology  Feb 2012 
 - 862-71 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1525-2191 
<br/><br/><label>Standard No.: </label>10.1016/j.ajpath.2011.10.018  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cell Movement--radiation effects<br/>Female<br/>Lung Neoplasms--secondary<br/>MAP Kinase Signaling System--radiation effects<br/>Mice<br/>Mice, Inbred BALB C<br/>Mice, Nude<br/>Neoplasm Metastasis<br/>Neoplasm Recurrence, Local--metabolism<br/>Neoplasms, Radiation-Induced--metabolism<br/>Phosphorylation<br/>Proto-Oncogene Proteins c-raf--metabolism<br/>Radiation Tolerance--radiation effects<br/>Tumor Cells, Cultured<br/>Uterine Cervical Neoplasms--metabolism<br/>Wound Healing--radiation effects<br/>p38 Mitogen-Activated Protein Kinases--physiology<br/>ras Proteins--genetics