Hu, Jinsong <br/><br/>
Activation of ATF4 mediates unwanted Mcl-1 accumulation by proteasome inhibition.  [electronic resource] 

 -  Blood  Jan 2012 
 - 826-37 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1528-0020 
<br/><br/><label>Standard No.: </label>10.1182/blood-2011-07-366492  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Activating Transcription Factor 4--genetics<br/>Antineoplastic Agents--pharmacology<br/>Apoptosis<br/>Blotting, Western<br/>Boronic Acids--pharmacology<br/>Bortezomib<br/>Cell Line, Tumor<br/>Cell Proliferation<br/>Chromatin Immunoprecipitation<br/>DNA-Binding Proteins--antagonists & inhibitors<br/>Endoplasmic Reticulum--metabolism<br/>Endoplasmic Reticulum Stress--drug effects<br/>Gene Expression Regulation, Neoplastic<br/>Humans<br/>Multiple Myeloma--drug therapy<br/>Myeloid Cell Leukemia Sequence 1 Protein<br/>Proto-Oncogene Proteins c-bcl-2--genetics<br/>Pyrazines--pharmacology<br/>RNA Splicing<br/>RNA, Messenger--genetics<br/>RNA, Small Interfering--genetics<br/>Real-Time Polymerase Chain Reaction<br/>Regulatory Factor X Transcription Factors<br/>Transcription Factors--antagonists & inhibitors<br/>Transcription, Genetic<br/>Unfolded Protein Response--drug effects