Kubin, Thomas <br/><br/>
Oncostatin M is a major mediator of cardiomyocyte dedifferentiation and remodeling.  [electronic resource] 

 -  Cell stem cell  Nov 2011 
 - 420-32 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1875-9777 
<br/><br/><label>Standard No.: </label>10.1016/j.stem.2011.08.013  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Biomarkers--metabolism<br/>Blotting, Western<br/>Cardiomyopathy, Dilated--metabolism<br/>Cardiotonic Agents--metabolism<br/>Cell Cycle--drug effects<br/>Cell Dedifferentiation--drug effects<br/>DNA--biosynthesis<br/>Fluorescent Antibody Technique<br/>Gene Deletion<br/>Gene Expression Regulation--drug effects<br/>Heart Function Tests--drug effects<br/>Humans<br/>Mice<br/>Mice, Transgenic<br/>Myocardial Infarction--metabolism<br/>Myocardium--pathology<br/>Myocytes, Cardiac--drug effects<br/>Oncostatin M--metabolism<br/>Oncostatin M Receptor beta Subunit--metabolism<br/>Rats<br/>Signal Transduction--drug effects<br/>Stem Cells--cytology<br/>Ventricular Remodeling--drug effects