Voets, A M <br/><br/>
Transcriptional changes in OXPHOS complex I deficiency are related to anti-oxidant pathways and could explain the disturbed calcium homeostasis.  [electronic resource] 

 -  Biochimica et biophysica acta  Jul 2012 
 - 1161-8 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>0006-3002 
<br/><br/><label>Standard No.: </label>10.1016/j.bbadis.2011.10.009  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Antioxidants--metabolism<br/>Calcium--metabolism<br/>Cell Cycle Proteins--metabolism<br/>Child, Preschool<br/>DNA Damage<br/>Electron Transport Complex I--deficiency<br/>Endoplasmic Reticulum Stress<br/>Female<br/>Fibroblasts--cytology<br/>Gene Expression Profiling<br/>Glutathione--metabolism<br/>Homeostasis--genetics<br/>Humans<br/>Infant<br/>Infant, Newborn<br/>Male<br/>Metabolic Networks and Pathways--genetics<br/>Mitochondrial Diseases--genetics<br/>NF-E2-Related Factor 2--metabolism<br/>Nuclear Proteins--metabolism<br/>Oxidative Phosphorylation<br/>Oxidative Stress<br/>Selenoproteins--metabolism