Brambilla, Roberta <br/><br/>
Inhibition of soluble tumour necrosis factor is therapeutic in experimental autoimmune encephalomyelitis and promotes axon preservation and remyelination.  [electronic resource] 

 -  Brain : a journal of neurology  Sep 2011 
 - 2736-54 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural 
<br/><br/><label>ISSN: </label>1460-2156 
<br/><br/><label>Standard No.: </label>10.1093/brain/awr199  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Axons--immunology<br/>Chemokines--immunology<br/>Cytokines--immunology<br/>Encephalomyelitis, Autoimmune, Experimental--immunology<br/>Female<br/>Glycoproteins--immunology<br/>Humans<br/>Leukocytes--cytology<br/>Mice<br/>Mice, Inbred C57BL<br/>Multiple Sclerosis--immunology<br/>Myelin Sheath--metabolism<br/>Myelin-Oligodendrocyte Glycoprotein<br/>Nerve Regeneration--physiology<br/>Peptide Fragments--immunology<br/>Receptors, Tumor Necrosis Factor--metabolism<br/>Spinal Cord--cytology<br/>Tumor Necrosis Factor Inhibitors<br/>Tumor Necrosis Factors--metabolism