Deletion of UCP2 in iNOS deficient mice reduces the severity of the disease during experimental autoimmune encephalomyelitis. [electronic resource]
- PloS one 2011
- e22841 p. digital
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
1932-6203
10.1371/journal.pone.0022841 doi
Animals Brain--metabolism Cytokines--genetics Encephalomyelitis, Autoimmune, Experimental--genetics Glutathione--metabolism Glutathione Disulfide--metabolism Humans Immune System--metabolism Ion Channels--deficiency Macrophages, Peritoneal--metabolism Mice Mice, Knockout Mitochondrial Proteins--deficiency Nitric Oxide--metabolism Nitric Oxide Synthase Type II--deficiency Oxidation-Reduction Oxidative Stress Reactive Oxygen Species--metabolism Reverse Transcriptase Polymerase Chain Reaction Severity of Illness Index Spinal Cord--metabolism Uncoupling Protein 2