Al-Mulla, Fahd <br/><br/>
Impaired TGF-β signaling and a defect in resolution of inflammation contribute to delayed wound healing in a female rat model of type 2 diabetes.  [electronic resource] 

 -  Molecular bioSystems  Nov 2011 
 - 3006-20 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1742-2051 
<br/><br/><label>Standard No.: </label>10.1039/c0mb00317d  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Cell Line<br/>Cell Movement<br/>Diabetes Mellitus, Type 2--immunology<br/>Estrogens--metabolism<br/>Female<br/>Fibroblasts--pathology<br/>Humans<br/>Inflammation--metabolism<br/>Macrophages--pathology<br/>NF-kappa B--physiology<br/>Oxidative Stress<br/>Rats<br/>Rats, Wistar<br/>Signal Transduction<br/>Skin--cytology<br/>Smad2 Protein--metabolism<br/>Smad7 Protein--metabolism<br/>Transforming Growth Factor beta--metabolism<br/>Tumor Necrosis Factor-alpha--physiology<br/>Wound Healing