Farooq, Faraz <br/><br/>
Prolactin increases SMN expression and survival in a mouse model of severe spinal muscular atrophy via the STAT5 pathway.  [electronic resource] 

 -  The Journal of clinical investigation  Aug 2011 
 - 3042-50 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1558-8238 
<br/><br/><label>Standard No.: </label>10.1172/JCI46276  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Disease Models, Animal<br/>Endothelial Cells--cytology<br/>Gene Expression Regulation<br/>Humans<br/>Mice<br/>Motor Neurons--physiology<br/>Muscular Atrophy, Spinal--metabolism<br/>Neurons--metabolism<br/>Prolactin--biosynthesis<br/>RNA, Messenger--metabolism<br/>STAT5 Transcription Factor--metabolism<br/>Survival of Motor Neuron 1 Protein--biosynthesis<br/>Transcription, Genetic<br/>Tumor Suppressor Proteins--metabolism<br/>Up-Regulation