Delayed neuroprotection induced by sevoflurane via opening mitochondrial ATP-sensitive potassium channels and p38 MAPK phosphorylation. [electronic resource]
- Neurological sciences : official journal of the Italian Neurological Society and of the Italian Society of Clinical Neurophysiology Apr 2012
- 239-49 p. digital
Publication Type: Journal Article
1590-3478
10.1007/s10072-011-0665-6 doi
Analysis of Variance Animals Anti-Arrhythmia Agents--pharmacology Brain Infarction--etiology Cell Count Decanoic Acids--pharmacology Disease Models, Animal Gene Expression Regulation--drug effects Hydroxy Acids--pharmacology Infarction, Middle Cerebral Artery--drug therapy Ischemic Preconditioning--methods Male Methyl Ethers--therapeutic use Neuroprotective Agents--therapeutic use Phosphorylation--drug effects Potassium Channels--metabolism Rats Rats, Sprague-Dawley Sevoflurane Time Factors p38 Mitogen-Activated Protein Kinases--metabolism