Cao, Wei <br/><br/>
Burn-induced apoptosis of cardiomyocytes is survivin dependent and regulated by PI3K/Akt, p38 MAPK and ERK pathways.  [electronic resource] 

 -  Basic research in cardiology  Nov 2011 
 - 1207-20 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, Non-U.S. Gov't 
<br/><br/><label>ISSN: </label>1435-1803 
<br/><br/><label>Standard No.: </label>10.1007/s00395-011-0199-3  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Animals<br/>Apoptosis--physiology<br/>Blotting, Western<br/>Burns--metabolism<br/>Cytokines--blood<br/>Disease Models, Animal<br/>Enzyme-Linked Immunosorbent Assay<br/>Heart Ventricles--metabolism<br/>Immunohistochemistry<br/>In Situ Nick-End Labeling<br/>MAP Kinase Signaling System--physiology<br/>Male<br/>Microtubule-Associated Proteins--metabolism<br/>Myocytes, Cardiac--metabolism<br/>Phosphatidylinositol 3-Kinases--metabolism<br/>Proto-Oncogene Proteins c-akt--metabolism<br/>RNA, Small Interfering<br/>Rats<br/>Rats, Sprague-Dawley<br/>Real-Time Polymerase Chain Reaction<br/>Signal Transduction--physiology<br/>Survivin<br/>Transfection<br/>p38 Mitogen-Activated Protein Kinases--metabolism