Kelley, Joshua B <br/><br/>
The defective nuclear lamina in Hutchinson-gilford progeria syndrome disrupts the nucleocytoplasmic Ran gradient and inhibits nuclear localization of Ubc9.  [electronic resource] 

 -  Molecular and cellular biology  Aug 2011 
 - 3378-95 p.  digital 
<br/><br/> Publication Type: Journal Article; Research Support, N.I.H., Extramural; Research Support, Non-U.S. Gov't; Research Support, U.S. Gov't, Non-P.H.S. 
<br/><br/><label>ISSN: </label>1098-5549 
<br/><br/><label>Standard No.: </label>10.1128/MCB.05087-11  doi 
<br/><br/><label>Subjects--Topical Terms: </label><br/>Active Transport, Cell Nucleus<br/>Cell Cycle Proteins<br/>Guanine Nucleotide Exchange Factors<br/>Humans<br/>Nuclear Lamina--pathology<br/>Nuclear Proteins<br/>Progeria--metabolism<br/>Sumoylation<br/>Ubiquitin-Conjugating Enzymes--antagonists & inhibitors<br/>ran GTP-Binding Protein--metabolism<br/>Ubiquitin-Conjugating Enzyme UBC9